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Conclusions: Gastric acid plays a role in suppressing the proximal propagation of H. pylori-induced gastritis to the bodyofstomach.
1
Seventy-two hours later, gastriccorpus tissues were taken for quantitative histological assessment.
2
Apoptosis was also significantly increased in gastriccorpus biopsies obtained from H. pylori-infected humans with moderate degrees of hypergastrinemia.
3
Design: Epithelial cells were isolated from the gastriccorpus of healthy stomachs and stomachs with drug-induced and inflammation-induced SPEM lesions.
4
The nitric oxide synthase (NOS) activity was measured in the surface and deep mucosa of the gastriccorpus.
5
We conclude that hypergastrinemia specifically renders cells within the gastriccorpus epithelium more susceptible to induction of apoptosis by radiation or H. pylori.
Usage of gastric body in English
1
CT failed to demonstrate two fundal and 19 antral or gastricbody lesions.
2
Results: In the gastricbody, the densities of H. pylori were not different among groups.
3
Upper gastrointestinal endoscopy revealed irregularly elevated tumors in the lower anterior gastricbody, which were diagnosed to be metastasis from HCC.
4
Gastroendoscopic findings showed an 8.0-cm submucosal tumor with ulcer on the greater curvature of the gastricbody.
5
The upper posterior gastricbody could be the most feasible location for performing TU-ESD, perhaps because of the difference in the subcutaneous dissection time.